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<table border="0" cellpadding="3" cellspacing="1" width="100%" class="forumline"> <tr> <th class="thCornerL" width="22%" height="26">Autor</th> <th class="thCornerR">Wiadomość</th> </tr> <tr> <td width="22%" align="left" valign="top" class="row1"><span class="name"><a name=""></a><b>yxgaxjlywf</b></span></td> <td class="row1" height="28" valign="top"><table width="100%" border="0" cellspacing="0" cellpadding="0"> <tr> <td width="100%"><img src="http://picsrv.fora.pl/xeon/images/icon_minipost.gif" width="12" height="9" alt="Post" title="Post" border="0" /><span class="postdetails">Wysłany: Pon 16:10, 07 Mar 2011<span class="gen"> </span> Temat postu: tory burch reva dtd lug gmj onx</span></td> </tr> <tr> <td colspan="2"><hr /></td> </tr> <tr> <td colspan="2"><span class="postbody">Captopril of bradykinin on neonatal rat cardiac fibroblasts inhibit the proliferation of cells <br /> <br /> <br />Part of the role can be antagonistic AnglI:. Recent studies have reported that ACEI is B receptor agonists, can promote JCiinCardioI (China),<a href="http://www.toryburchreva.net" target="_blank" class="postlink">tory burch reva</a>, Sep2005. Vol21, No9B receptor expression: thus, receptors may be partially mediated the role of ACEI. Ang Ⅱ receptor in turn activated by AT BK / NO pathway,<a href="http://www.ghd-italia.net" target="_blank" class="postlink">ghd italia</a>, inhibit the development of myocardial fibrosis:, suggesting that NO may be anti-cardiac hypertrophy and myocardial fibrosis plays an important role. NO signal transduction pathway through to generate a negative growth factor in the role of cGMP,<a href="http://www.mbtshoesitalia.net" target="_blank" class="postlink">mbt shoes italia</a>, which the proliferation of many cells with negative regulatory role. In addition, cGMP may be the BK / NO inhibited the proliferation of CFs part of a cell regulator. CFs in the regulation of proliferation and play an important role:. This study demonstrates that blocking BK by HOE 140, the role of a, the cell culture supernatants and intracellular cGMP No content was significantly lower, indicating that BK-mediated proliferative effect of captopril against CFs generated with NO and cGMP on , which Kim et al: Study conclusions are consistent. In summary, captopril can inhibit the proliferation of CFs,<a href="http://www.toryburchshoesoutlet.net" target="_blank" class="postlink">tory burch outlet</a>, the development of cardiac hypertrophy have some reverse effect,<a href="http://www.scarpe-adidas.net" target="_blank" class="postlink">adidas scarpe</a>, which might in part by the BK receptor-mediated by the J3, and with the BK / NO / cGMP signaling transduction pathways involved in regulation. However, the specific regulatory mechanisms remains to be further studied.</span></td> </tr> </table></td> </tr> <tr> <td colspan="2" height="1" class="spaceRow"><img src="http://picsrv.fora.pl/xeon/images/spacer.gif" alt="" width="1" height="1" /></td> </tr> </table>
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