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<table border="0" cellpadding="3" cellspacing="1" width="100%" class="forumline"> <tr> <th class="thCornerL" width="22%" height="26">Autor</th> <th class="thCornerR">Wiadomość</th> </tr> <tr> <td width="22%" align="left" valign="top" class="row1"><span class="name"><a name=""></a><b>yxgaxjlywf</b></span></td> <td class="row1" height="28" valign="top"><table width="100%" border="0" cellspacing="0" cellpadding="0"> <tr> <td width="100%"><img src="http://picsrv.fora.pl/xeon/images/icon_minipost.gif" width="12" height="9" alt="Post" title="Post" border="0" /><span class="postdetails">Wysłany: Pią 18:02, 25 Mar 2011<span class="gen"> </span> Temat postu: 56 cases of cerebral thrombosis Clinical and CT an</span></td> </tr> <tr> <td colspan="2"><hr /></td> </tr> <tr> <td colspan="2"><span class="postbody">56 cases of cerebral thrombosis Clinical and CT analysis <br /> <br /> <br />56 cases of limb paralysis in the lesion size and a certain degree of relationship between the lesions more than 4.0cm in 4 cases of severe paralysis moderate in 3 cases, mild in 2 cases; lesions in the 1.5 ~ 4.0cm, 5 cases of severe paralysis, moderate 8 cases of paralysis, mild paralysis in 5 cases, 1 case of non-limb paralysis; lesions 1.5cm below the 3 cases of severe paralysis, paralysis of 8 patients with moderate and mild cases of paralysis l5; no exception and mild paralysis in 2 cases; l cases of brain atrophy paresis were 3 cases of them sicker after the review of CT lesions expand, l cases in the right basal ganglia lesions extend from the 0.8cm × 0.8cm to 1.4cm × 0.5cm; l cases in Ministry of the left temporal lobe extended to the 3.3cm × 1.9cm 3.5cm × 3.0cm; l l cases in the Department of the pons from the exception that there was no lesion 0.8cm × 0.9cm. Another change in 4 cases with special conditions,<a href="http://www.toryburchshoesoutlets.com" target="_blank" class="postlink">tory burch shoes</a>, l cases of CT after admission confirmed by the right frontal lobe, lenticular nucleus, caudate between the cerebral infarction. 17d in the hospital after the sudden consciousness, deep coma at the dry state, the right pupil dilated, lumbar puncture and cerebrospinal fluid for the turbid pale yellow liquid, Pan's response to H, cells microscope 6/dl, fresh red blood cells scattered in the full view (excluding wrong to wear.) Biochemical protein 1.9g / L, glucose: 4.7mmol / L, HG 214.6mmol / L. He died. Hemorrhagic infarction and brain herniation due to death. L other patients on admission have severe headache, nausea and vomiting. Blood pressure 32/17kPa. Ming Qing Language of God, limb muscle strength, muscle tone normal, hypertension was diagnosed with stage Ⅲ, hypertensive encephalopathy, with antihypertensive drugs and the effect of mannitol. Illness 9dCT check for multiple brain ridge plug. Hypertension past i0 years. Multiple small lesions or the occurrence of annoying and the quiet zone, so the signs were not obvious. The other 2 cases were clinically mild limb Yao. 30h disease and 2 months later CT revealed no abnormalities, possibly infarct CT examination is an important means of diagnosis of cerebrovascular disease, but also an important basis for the treatment prognosis. In the acute phase of cerebral blood vessels, especially in severe cases, edema, do not immediately use vasodilators. The security of blood syndrome. CT diagnosis of low-density area on the edge of space occupying unclear responders, first with the dehydration treatment, and then use the vasodilator drugs, this treatment achieved satisfactory clinical results.</span></td> </tr> </table></td> </tr> <tr> <td colspan="2" height="1" class="spaceRow"><img src="http://picsrv.fora.pl/xeon/images/spacer.gif" alt="" width="1" height="1" /></td> </tr> </table>
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